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Альманах клинической медицины. 2014; : 3-11

ОСОБЕННОСТИ НЕФРОПАТИЙ ПРИ ЛИМФОПРОЛИФЕРАТИВНЫХ И ПЛАЗМОКЛЕТОЧНЫХ ЗАБОЛЕВАНИЯХ (ОПЫТ ОДНОГО ЦЕНТРА)

Захарова Е. В., Столяревич Е. С.

https://doi.org/10.18786/2072-0505-2014-30-3-11

Аннотация

Проанализированы клинические данные 253 нефрологических больных, у которых диагностированы лимфопролиферативные и плазмоклеточные заболевания, и клинико-морфологические данные 128 из этих пациентов, у которых поражение почек верифицировано морфологически. Выявлен широкий спектр морфологических вариантов и клинико-морфологических корреляций.
Список литературы

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9. Heher E.C., Goes N.B., Spitzer T.R. et al. Kidney disease associated with plasma cell dyscrasias//Blood. 2010. V.116. P.1397-1404.

10. Herrera G.A., Joseph L., Gu X. et al. Renal pathological spectrum in an autopsy series of patients with plasma cell dyscrasia//Arch. Path. Lab. Med. 2004. V.128. P.875-879.

11. Keeling J., Teng J., Herrera G.A. AL-amyloidosis and light-chain deposition disease: light chains induce divergent phenotypic transformations of human mesangial cells//Lab. Invest. 2004. V.84, No.10. P.1322-1338.

12. Korbet S.M., Schwartz M.M. Multiple myeloma//J. Am. Soc. Nephrol. 2006. V.17. P.2533-2545.

13. Leung N., Bridoux F., Hutchinson С.A. et al. Monoclonal gammopathy of renal significance (MGRS): when MGUS is no longer undetermined or insignificant//Blood. 2012. V.120, No.22. P.4292-4295.

14. Leung N., Dispenzieri A., Fervenza F.C. et al. Renal response after high-dose melphalan and stem cell transplantation is a favorable marker in patients with primary systemic amyloidosis//Am. J. Kidney Dis. 2005. V.46, No.2. P.270-277.

15. Nasr S.H., Markowitz G.S., Stokes M.B. et al. Proliferative glomerulonephritis with monoclonal IgG deposits; a distinct entity mimicking immune-complex glomerulonephritis//Kidney Int. 2004. V.65. P.85-96.

16. Ronco P.M. Paraneoplastic glomerulopathies: new insights into an old entity 11 Kidney Int. 1999. V.56. P.355-377.

17. Ronco P., Plaisier E., Aucouturier P. Ig-related renal disease in lymphoplasmacytic disorders: An update//Semin. Nephrol. 2010. V.30. P.557-569.

18. Royer B., Arnulf В., Martinez F. et al. High dose chemotherapy in light chain or light and heavy chain deposition disease//Kidney Int. 2004. V.65. P.642-648.

19. Sanders P.W., Booker B.B. Pathobiology of cast nephropathy from human Bence-Jones proteins//J. Clin. Invest. 1992. V.89. P.630-639.

20. Weichman К., Dember L.M., Prokaeva T. at el. Clinical and molecular characteristics of patients with non-amyloid light chain deposition disorders, and outcome following treatment with high dose melphalan and autologous stem cell transplantation//Bone Marrow Transplantation. 2006. V.38. P.339-346.

Almanac of Clinical Medicine. 2014; : 3-11

KIDNEY DAMAGE IN LYMPHOPROLIFERATIVE AND PLASMA CELL DISORDERS (ONE CENTER EXPERIENCE)

Zakharova E. V., Stolyarevich E. S.

https://doi.org/10.18786/2072-0505-2014-30-3-11

Abstract

We analyzed clinical data of 253 patients with lymphoproliferative and plasma cell disorders and kidney damage, and clinical and pathological data of 128 of these patients with histologically proven nephropathy. Broad spectrum of histological variants was found, and correlations between clinical diagnosis and pathology established.
References

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5. Cambier J.F., Ronco P. Onco-nephrology: glomerular diseases with cancer//Clin. J. Am. Soc. Nephrol. 2012. V.7, No.10. P.1701-1712.

6. Cohen A.H. The kidney in plasma cell dyscrasias: Bence-Jones cast nephropathy and light chain deposition disease//Am. J. Kidney Dis. 1998. V.32. P.529-532.

7. Da'as N., Polliack A., Cohen Y. et al. Kidney involvement and renal manifestations in non-Hodgkin's lymphoma and lymphocytic leukemia: a retrospective study in 700 patients//Eur. J. Haematol. 2001. V.67. P.158-160.

8. Magier K.T., Lynch J.W. Jr. Paraneoplastic manifestations of lymphoma//Clin. Lymphoma. 2004. V.5, No.l. P.29-36.

9. Heher E.C., Goes N.B., Spitzer T.R. et al. Kidney disease associated with plasma cell dyscrasias//Blood. 2010. V.116. P.1397-1404.

10. Herrera G.A., Joseph L., Gu X. et al. Renal pathological spectrum in an autopsy series of patients with plasma cell dyscrasia//Arch. Path. Lab. Med. 2004. V.128. P.875-879.

11. Keeling J., Teng J., Herrera G.A. AL-amyloidosis and light-chain deposition disease: light chains induce divergent phenotypic transformations of human mesangial cells//Lab. Invest. 2004. V.84, No.10. P.1322-1338.

12. Korbet S.M., Schwartz M.M. Multiple myeloma//J. Am. Soc. Nephrol. 2006. V.17. P.2533-2545.

13. Leung N., Bridoux F., Hutchinson S.A. et al. Monoclonal gammopathy of renal significance (MGRS): when MGUS is no longer undetermined or insignificant//Blood. 2012. V.120, No.22. P.4292-4295.

14. Leung N., Dispenzieri A., Fervenza F.C. et al. Renal response after high-dose melphalan and stem cell transplantation is a favorable marker in patients with primary systemic amyloidosis//Am. J. Kidney Dis. 2005. V.46, No.2. P.270-277.

15. Nasr S.H., Markowitz G.S., Stokes M.B. et al. Proliferative glomerulonephritis with monoclonal IgG deposits; a distinct entity mimicking immune-complex glomerulonephritis//Kidney Int. 2004. V.65. P.85-96.

16. Ronco P.M. Paraneoplastic glomerulopathies: new insights into an old entity 11 Kidney Int. 1999. V.56. P.355-377.

17. Ronco P., Plaisier E., Aucouturier P. Ig-related renal disease in lymphoplasmacytic disorders: An update//Semin. Nephrol. 2010. V.30. P.557-569.

18. Royer B., Arnulf V., Martinez F. et al. High dose chemotherapy in light chain or light and heavy chain deposition disease//Kidney Int. 2004. V.65. P.642-648.

19. Sanders P.W., Booker B.B. Pathobiology of cast nephropathy from human Bence-Jones proteins//J. Clin. Invest. 1992. V.89. P.630-639.

20. Weichman K., Dember L.M., Prokaeva T. at el. Clinical and molecular characteristics of patients with non-amyloid light chain deposition disorders, and outcome following treatment with high dose melphalan and autologous stem cell transplantation//Bone Marrow Transplantation. 2006. V.38. P.339-346.