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Валеология: Здоровье, Болезнь, Выздоровление. 2020; : 85-88

СОСТОЯНИЕ ЛЕВОГО ЖЕЛУДОЧКА У ПАЦИЕНТОВ С ИШЕМИЧЕСКОЙ БОЛЕЗНЬЮ СЕРДЦА

КУБЕКОВА С. Ж., ЖУСУПОВА Г. К., БИКТАШЕВ Д. Б., СМАИЛОВА Г. Т., ЗАГОРУЛЯ Н. Л., РИБ Е. А.

Аннотация

   В настоящей статье проведен обзор литературы, затрагивающий одну из самых распространенных проблем современной кардиологии – состояние левых отделов сердца при наиболее часто встречающемся заболевании – ишемической болезни сердца.

Список литературы

1. Tadic M., Pieske-Kraigher E., Cuspidi C. et al. Left ventricular strain and twisting in heart failure with preserved ejection fraction: an updated review // Heart Fail Rev. – 2017. – Vol. 22, № 3. – P. 371-379.

2. Lauritsen J., Gustafsson F., Abdulla J. Characteristics and long‐term prognosis of patients with heart failure and mid‐range ejection fraction compared with reduced and preserved ejection fraction: a systematic review and meta‐analysis // ESC Heart Fail. – 2018. – Vol. 5, № 4. – P. 385-394.

3. Owen B., Bojdo N., Jivkov A. et al. Structural modelling of the cardiovascular system // Biomech Model Mechanobiol. – 2018. – Vol. 17, № 5. – P. 1217-1242.

4. Packer M. The neurohormonal hypothesis: a theory to explain the mechanism of disease progression in heart failure // J Am Coll Cardiol. – 1992. – Vol. 20, № 1. – P. 248-254.

5. Benedict C. R., Johnstone D. E., Weiner D. H. et al. Relation of neurohumoral activation to clinical variables and degree of ventricular dysfunction: a report from the Registry of Studies of Left Ventricular Dysfunction // J Am Coll Cardiol. – 1994. – Vol. 23, № 6. – P. 1410-1420.

6. Karnik S., Unal H., Kemp J. et al. International Union of Basic and Clinical Pharmacology. XCIX. Angiotensin Receptors: Interpreters of Pathophysiological Angiotensinergic Stimuli // Pharmacol Rev. – 2015. – Vol. 67, № 4. – P. 754-819.

7. Leask A. Getting to the heart of the matter: new insights into cardiac fibrosis // Circ Res. – 2015. – Vol. 116, № 7. – P. 1269-1276.

8. Hartupee J., Mann D. Neurohormonal activation in heart failure with reduced ejection fraction // Nat Rev Cardiol. – 2017. – Vol. 14, № 1. – P. 30-38.

9. Sparks M., Crowley S., Gurley S. et al. Classical Renin-Angiotensin System in Kidney Physiology // Compr Physiol. – 2014. – Vol. 4, №3. – P. 1201-1228.

10. Zablocki D., Sadoshima J. Angiotensin II and Oxidative Stress in the Failing Heart // Antioxid Redox Signal. – 2013. – Vol. 19, № 10. – P. 1095-1109.

11. Lijnen P., Petrov V. Induction of cardiac fibrosis by aldosterone // J Mol Cell Cardiol. – 2000. – Vol. 32, № 6. – P. 865-879.

12. Brown J., Underwood P., ferri C. et al. Aldosterone dysregulationwith aging predicts renal-vascular-function and cardio-vascular risk // Hypertension. – 2014. – Vol. 63, № 6. – P. 1205-1211.

13. Libby P., Theroux P. Pathophysiology of Coronary Artery Disease // Circulation. – 2005. – Vol. 111, № 25. – P. 3481-3488.

14. Libby P. Inflammation in atherosclerosis // Arteriosclerosis, Thrombosis and, Vascular Biology. – 2012. – Vol. 32, № 9. – P. 2045-2051.

15. Henry L. B. Left Ventricular Systolic Dysfunction and Ischemic Cardiomyopathy // Crit Care Nurs Q. Journal. – 2003. – Vol. 26, № 1. – P. 16-21.

16. Burchfield J., Xie M., Hill J. Pathological Ventricular Remodeling // Circulation. – 2013. – Vol. 128, №4. – P. 388-400.

17. Buglioni A., Burnett J. Pathophisiology and the cardiorenal connection in heart failure. Circulating hormones: biomarkers of mediators // Clin Chim Acta. – 2015. – Vol. 443. – P. 3-8.

18. Schwarz Е., Dashti R. The clinical quandary of left and right ventricular diastolic dysfunction and diastolic heart failure // Cardiovasc J Afr. – 2010. – Vol. 21, № 4. – P. 212-220.

19. Bière L., Donal E., Jacquier A. et al. A new look at left ventricular remodeling definition by cardiac imaging // Int J Cardiol. – 2016. – Vol. 209. – P. 17-19.

20. Lerman A., Holmes D. R., Herrmann J. et al. Microcirculatory dysfunction in ST-elevation myocardial infarction: cause, consequence, or both? // Eur Heart J. – 2007. – Vol. 28, № 7. – P. 788-797.

21. Prasad A., Stone G. W., Holmes D. R. et al. Reperfusion injury, microvascular dysfunction and cardioprotection: the «dark side» of reperfusion // Circulation. – 2009. – Vol. 120, № 21. – P. 2105-2112.

22. Castorena-Gonzalez J., Staiculescu M., Foote C. et al. Mechanisms of the inward remodeling process in resistance vessels: Is the actin cytoskeleton involved? // Microcirculation. –2014. – Vol. 21, № 3. – P. 219-229.

23. Bauters C., Dubois S., Porouchani S. et al. Long-term prognostic impact of left ventricular remodeling after a first myocardial infarction in modern clinical practice // PloS One. – 2017. – Vol.12, № 11. – P. e0188884.

24. Huttin O., Coiro S., Selton-Suty C. et al. Prediction of Left Ventricular Remodeling after a Myocardial Infarction: Role of Myocardial Deformation: A Systematic Review and Meta-Analysis // PLoS One. – 2016. – Vol. 11, № 12. – Р. e0168349.

25. Hendrics T., Hartman M., Vlaar P. et al. Predictors of left ventricular remodeling after ST-elevation myocardial infarction // Int J Cardiovasc Imaging. – 2017. – Vol. 33, № 9. – P. 1415-1423.

26. Mollema S., Liem S., Suffoletto M. et al. Left ventricular dyssynchrony acutely after myocardial infarction predicts left ventricular remodeling // J Am Coll Cardiol. – 2007. – Vol. 50, № 16. – P. 1532-1540.

27. Hellawell J. L., Margulies K. B. Myocardial reverse remodeling // Cardiovasc Ther. – 2012. – Vol. 30, № 3. – P. 172-181.

28. Daubert C., Gold M. R., Abraham W. T. et al. Prevention of disease progression by cardiac resynchronization therapy in patients with asymptomatic or mildly symptomatic left ventricular dysfunction: Insights from the European cohort of the REVERSE (Resynchronization Reverses Remodeling in Systolic Left Ventricular Dysfunction) trial // J Am Coll Cardiol. – 2009. – Vol. 54, № 20. – P. 1837-1846.

Valeology: Health - Illnes - recovery. 2020; : 85-88

STATE OF THE LEFT VENTRICLE IN PATIENTS WITH CORONARY HEART DISEASE

KUBEKOVA S. , ZHUSUPOVA G. , BIKTASHEV D. , SMAILOVA G. , ZAGORULYA N. , RIB E.

Abstract

   This article reviews the literature on one of the most common problems of modern cardiology - the state of the left heart in the most common disease – coronary heart disease.

References

1. Tadic M., Pieske-Kraigher E., Cuspidi C. et al. Left ventricular strain and twisting in heart failure with preserved ejection fraction: an updated review // Heart Fail Rev. – 2017. – Vol. 22, № 3. – P. 371-379.

2. Lauritsen J., Gustafsson F., Abdulla J. Characteristics and long‐term prognosis of patients with heart failure and mid‐range ejection fraction compared with reduced and preserved ejection fraction: a systematic review and meta‐analysis // ESC Heart Fail. – 2018. – Vol. 5, № 4. – P. 385-394.

3. Owen B., Bojdo N., Jivkov A. et al. Structural modelling of the cardiovascular system // Biomech Model Mechanobiol. – 2018. – Vol. 17, № 5. – P. 1217-1242.

4. Packer M. The neurohormonal hypothesis: a theory to explain the mechanism of disease progression in heart failure // J Am Coll Cardiol. – 1992. – Vol. 20, № 1. – P. 248-254.

5. Benedict C. R., Johnstone D. E., Weiner D. H. et al. Relation of neurohumoral activation to clinical variables and degree of ventricular dysfunction: a report from the Registry of Studies of Left Ventricular Dysfunction // J Am Coll Cardiol. – 1994. – Vol. 23, № 6. – P. 1410-1420.

6. Karnik S., Unal H., Kemp J. et al. International Union of Basic and Clinical Pharmacology. XCIX. Angiotensin Receptors: Interpreters of Pathophysiological Angiotensinergic Stimuli // Pharmacol Rev. – 2015. – Vol. 67, № 4. – P. 754-819.

7. Leask A. Getting to the heart of the matter: new insights into cardiac fibrosis // Circ Res. – 2015. – Vol. 116, № 7. – P. 1269-1276.

8. Hartupee J., Mann D. Neurohormonal activation in heart failure with reduced ejection fraction // Nat Rev Cardiol. – 2017. – Vol. 14, № 1. – P. 30-38.

9. Sparks M., Crowley S., Gurley S. et al. Classical Renin-Angiotensin System in Kidney Physiology // Compr Physiol. – 2014. – Vol. 4, №3. – P. 1201-1228.

10. Zablocki D., Sadoshima J. Angiotensin II and Oxidative Stress in the Failing Heart // Antioxid Redox Signal. – 2013. – Vol. 19, № 10. – P. 1095-1109.

11. Lijnen P., Petrov V. Induction of cardiac fibrosis by aldosterone // J Mol Cell Cardiol. – 2000. – Vol. 32, № 6. – P. 865-879.

12. Brown J., Underwood P., ferri C. et al. Aldosterone dysregulationwith aging predicts renal-vascular-function and cardio-vascular risk // Hypertension. – 2014. – Vol. 63, № 6. – P. 1205-1211.

13. Libby P., Theroux P. Pathophysiology of Coronary Artery Disease // Circulation. – 2005. – Vol. 111, № 25. – P. 3481-3488.

14. Libby P. Inflammation in atherosclerosis // Arteriosclerosis, Thrombosis and, Vascular Biology. – 2012. – Vol. 32, № 9. – P. 2045-2051.

15. Henry L. B. Left Ventricular Systolic Dysfunction and Ischemic Cardiomyopathy // Crit Care Nurs Q. Journal. – 2003. – Vol. 26, № 1. – P. 16-21.

16. Burchfield J., Xie M., Hill J. Pathological Ventricular Remodeling // Circulation. – 2013. – Vol. 128, №4. – P. 388-400.

17. Buglioni A., Burnett J. Pathophisiology and the cardiorenal connection in heart failure. Circulating hormones: biomarkers of mediators // Clin Chim Acta. – 2015. – Vol. 443. – P. 3-8.

18. Schwarz E., Dashti R. The clinical quandary of left and right ventricular diastolic dysfunction and diastolic heart failure // Cardiovasc J Afr. – 2010. – Vol. 21, № 4. – P. 212-220.

19. Bière L., Donal E., Jacquier A. et al. A new look at left ventricular remodeling definition by cardiac imaging // Int J Cardiol. – 2016. – Vol. 209. – P. 17-19.

20. Lerman A., Holmes D. R., Herrmann J. et al. Microcirculatory dysfunction in ST-elevation myocardial infarction: cause, consequence, or both? // Eur Heart J. – 2007. – Vol. 28, № 7. – P. 788-797.

21. Prasad A., Stone G. W., Holmes D. R. et al. Reperfusion injury, microvascular dysfunction and cardioprotection: the «dark side» of reperfusion // Circulation. – 2009. – Vol. 120, № 21. – P. 2105-2112.

22. Castorena-Gonzalez J., Staiculescu M., Foote C. et al. Mechanisms of the inward remodeling process in resistance vessels: Is the actin cytoskeleton involved? // Microcirculation. –2014. – Vol. 21, № 3. – P. 219-229.

23. Bauters C., Dubois S., Porouchani S. et al. Long-term prognostic impact of left ventricular remodeling after a first myocardial infarction in modern clinical practice // PloS One. – 2017. – Vol.12, № 11. – P. e0188884.

24. Huttin O., Coiro S., Selton-Suty C. et al. Prediction of Left Ventricular Remodeling after a Myocardial Infarction: Role of Myocardial Deformation: A Systematic Review and Meta-Analysis // PLoS One. – 2016. – Vol. 11, № 12. – R. e0168349.

25. Hendrics T., Hartman M., Vlaar P. et al. Predictors of left ventricular remodeling after ST-elevation myocardial infarction // Int J Cardiovasc Imaging. – 2017. – Vol. 33, № 9. – P. 1415-1423.

26. Mollema S., Liem S., Suffoletto M. et al. Left ventricular dyssynchrony acutely after myocardial infarction predicts left ventricular remodeling // J Am Coll Cardiol. – 2007. – Vol. 50, № 16. – P. 1532-1540.

27. Hellawell J. L., Margulies K. B. Myocardial reverse remodeling // Cardiovasc Ther. – 2012. – Vol. 30, № 3. – P. 172-181.

28. Daubert C., Gold M. R., Abraham W. T. et al. Prevention of disease progression by cardiac resynchronization therapy in patients with asymptomatic or mildly symptomatic left ventricular dysfunction: Insights from the European cohort of the REVERSE (Resynchronization Reverses Remodeling in Systolic Left Ventricular Dysfunction) trial // J Am Coll Cardiol. – 2009. – Vol. 54, № 20. – P. 1837-1846.